CCR5 protein coverage in the major T-cell surface.
CCR5蛋白质主要覆盖于T细胞表面。
This variability means that a small deletion on the CCR5 gene.
这种变异是指CCR5基因上一小段缺失。
HIV virus is usually attached to a protein called CCR5 into human cells.
HIV病毒通常是附着在名为CCR5蛋白质潜入人体细胞的。
CCR5 protein coverage in the major T-cell surface, is a receptor protein.
CCR5蛋白质主要覆盖于T细胞表面，是一种蛋白受体。
CCR5, one of the coreceptors of HIV?1, is a new target of anti-HIV therapy.
趋化因子受体CCR5是HIV?1 进入靶细胞的辅助受体，因而成为开发抗艾滋病药物的新靶点。
Therefore, T cells no CCR5 protein, HIV virus almost difficult to invade human cells.
因此，T细胞没有了CCR5蛋白质，HIV病毒几乎很难入侵人体细胞。
In addition, CCR5 and the CD4 receptor is also a component of HIV into human cells points.
此外，CCR5还与CD 4受体一道组成艾滋病病毒进入人体细胞的关卡。
Other researchers are experimenting with gene therapy to fully prevent the emergence of CCR5.
其他研究人员正在试验基因疗法来完全阻止CCR5的产生。
For the HIV virus, not the CCR5 protein t cells, almost difficult to infiltrate the invasion.
对于HIV病毒来说，没有了CCR5蛋白质的T细胞，几乎很难渗透入侵。
HIV virus is usually attached to the normal CCR5 protein on the cell and enters the human body.
HIV病毒通常是附着于正常的CCR5蛋白质之上而进入人体细胞的。
Results Compared to B6 WT group, B6 CCR5 KO group succumbed to acute GVHD at an accelerated rate.
结果与B6WT组比较，B6CCR5KO组小鼠以更快的速度死于急性移植物抗宿主疾病（GVHD）；
Without CCR5 for the HIV to rob, the patient's immune cells can effectively resist HIV and prevent infection.
如果没有CCR5对艾滋病毒的“抢劫”，病人的免疫细胞能有效地抵抗艾滋病毒并防止其感染发生。
Since the virus can't gain a foothold on cells that lack CCR5, people who have the mutation have natural protection.
如果细胞上没有足够的CCR 5, HIV病毒就无法在细胞上立足，所以有发生这种突变的人自然可以受到保护。
The mutation is known as CCR5 delta32 and is found in 1 percent to 3 percent of white populations of European descent.
这种CCR5delta32的基因突变在欧洲的白人中的存在概率为1%到3%。
Moreover, people found in HIV disease progression, CCR5-preferring viruses would transform into CXCR4-preferring ones.
人们还发现在HIV感染疾病进展中，存在嗜ccr5病毒株向嗜cxcr4毒株的转化。
Objective To investigate the role of CC chemokine receptor 5(CCR5) in the immunological pathogenesis of hepatitis B(HB). ?
目的研究细胞趋化因子受体5（CCR5）在乙型病毒性肝炎（HB）免疫发病机制中的作用。
HIV USES the CCR5 as a co-receptor (in addition to CD4 receptors) to latch on to and ultimately destroy immune system cells.
HIV利用CCR5为受体(除此之外还有CD 4型受体)来锁住并最终破坏免疫系统细胞。
Gene mutation makes human cells no longer produce normal CCR5 protein, HIV virus so it can not enter the human cell carrier.
基因突变使得人体细胞不再产生正常的CCR5蛋白质，HIV病毒因此也就没有了可以进入人体细胞的载体。
The latter experiments also identified the cells on which CCR1 and CCR5 expression is important for promoting liver fibrosis.
最近的实验也表明表面有CCR1和CCR5表达的细胞在促进肝纤维化中也有重要作用。
In the recent years, the research of HIV-1 infecting immunocytes by chemokine receptor CCR5 and CXCR4 was caused more attention.
近年来，HIV - 1在借助趋化因子受体(C CR 5,CXCR4)感染免疫细胞方面的研究引起了广泛关注。
The CCR5 ribozyme molecule stops the patient's white blood cells producing CCR5, a protein that HIV USES to get into host cells.
这种CCR5核酶分子能中止了病人的白血细胞产生CCR5，后者是艾滋病毒用于进入宿主细胞的一种蛋白质。
Objective: to study the relation of RANTES and its receptor (CCR5) expression in gastric cancer and in lymph nodes with cancer metastasis.
目的：探讨RANTES及其受体(CCR5)在胃癌和转移淋巴结组织中的表达及其与胃癌转移的关系。
This patient's own cells had the usual amount of the CCR5 receptor, and the strain of HIV in his blood was the type that used the receptor.
回到这个研究中，这个患者存在着正常的CCR5基因，同时他所感染的病毒是利用这个基因复制的HIV类。
All of these biological effects are performed by binding of ligands to receptors. Elsewhere, CXCR3 and CCR5 are mark chemokine receptors in RA.
它们的这些生物学功能是通过配体与受体结合的方式来发挥的，而CXCR3与CCR5是RA中趋化因子的标志性受体。
Furthermore, expression of both CCR2 and CCR5 genes was enhanced significantly in activated macrophages when compared with non-activated macrophages.
而且，与 未激活的巨噬细胞比较，已经 激活的巨噬细胞CCR2，CCR5基因的表达增强更为明显。
CCR5, a membrane protein on cell surface, is a member of the G protein-coupled receptor superfamily and one of the major co-receptors for HIV-1infection.
CCR5为细胞膜蛋白，属于G蛋白偶联受体家族的成员，是HIV - 1入侵机体细胞的主要辅助受体之一。
Conclusion The recombinant adenovirus carrying antisense RNA to CCR5 has laid down a good foundation for studying its inhibiting effect on HIV-1 infection.
结论成功地构建了携带CCR5反义rna重组腺病毒载体，为研究其抗hiv 1的作用打下基础。
Conclusion The recombinant adenovirus carrying antisense RNA to CCR5 has laid down a good foundation for studying its inhibiting effect on HIV-1 infection.
结论成功地构建了携带CCR5反义rna重组腺病毒载体，为研究其抗hiv 1的作用打下基础。
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