CMV的 Sat-RNA是一种亚病毒剂，取决于其辅助病毒的复制和细胞间的扩散。
Sat-RNA of CMV is a subviral agent, which depends on its helper virus to replicate and cell to cell diffuse.
实际上，它可以增强病毒的复制。
病毒的复制呈指数增长过程，需朊病毒结合因子参与。
The prion replication is a process of exponential increase, which needs some binding factors of prion help.
另外，动物实验显示NKT细胞可致肝组织损伤并能抑制肝炎病毒的复制。
In addition, animal experiments show that NKT cells could induce hepatocyte injury and inhibit hepatitis virus replication.
在抗病毒植物基因工程中，利用病毒的复制酶基因是一种很有前途的方法。
It was a kind of promising method to use viral replicase gene in anti-virus plant genetic engineering.
抑制病毒的复制，可以使免疫系统在受到感染后进行自我修复，也可以增加T细胞数量。
Preventing the virus from replicating can help the immune system recover from the HIV infection and improve T-cell counts.
细小病毒的复制紧紧依赖于和增殖及分化有关的细胞因子，尤其是和细胞周期S-期有关的因子。
The replication of parvovirus is strongly dependent on cellular factors associated with proliferation and differentiation, especially those related to the S-phase of the cell cycle.
据大量的研究报道，在疱疹病毒的复制起源点附近有难得的回文，近距离重复，和反向重复结构。
A number of studies reported there are unusual clusters of palindromes, closely spacedrepeats and inverted repeats in the neighborhood of the replication origins of herpesviruses.
本文还提出了N蛋白的酶加工现象在弹状病毒的复制和转录的调控过程中可能起重要作用的设想。
The possible role of the proteolytic processing of the N-protein in the regulatory mechanism of both viral replication and transcription processes was discussed.
接受这种疗法的病人如果在感染的最初几个月同时服用三种药物，最能有效地抑制病毒的复制扩散。
Accept this kind of therapy if patients infected at the first few months of taking three drugs at the same time, the most effective in inhibiting the proliferation of viral replication.
在人体组织和实验鼠身上进行的初步试验显示，其中的一种物质可在一段时期内使艾滋病病毒的复制水平降低98%。
In human tissues and mice was carried out in the preliminary tests have shown, in which a substance can be for a period of time so that the level of HIV replication to reduce 98%.
Fauci说：“它帮助证明了一个概念，即如果基因治疗能阻断CCR5的表达，那就可能会抑制病毒的复制能力。”
"It helps prove the concept that if somehow you can block the expression of CCR5, maybe by gene therapy, you might be able to inhibit the ability of the virus to replicate," Fauci said.
然而，研究结果发现，在动物模型以及艾滋病感染中，CD 8 +T细胞亚类细胞仍然能在很大程度上抑制病毒的复制。
However, research findings showed that a subset of CD8 + t cells was largely able to curtail viral replication in animal models and HIV infections.
干扰素刺激基因所编码干扰素刺激蛋白的高水平表达可以抑制病毒的复制，这种抑制是导致慢病毒建立潜伏感染的原因之一。
The over expression of interferon stimulated protein can inhibit the replication of viruses. The inhibition can lend lentivirus to establish latent infection.
通过TGEV分别感染IBRS2和IBRS2抗性细胞产生的病变，结果表明该反义RNA对病毒的复制有抑制作用，其抑制率近50%。
Antiviral activitity of the antisense RNA was evaluated by cytopathic effect using cultured IBRS2 cell and anti- IBRS2 cell infected by TGEV respectively, and the inhibitory rate was about 50%.
他认为Nagot博士等撰写的文章对临床治疗有直接的指导作用，提示HIV - 1病毒的复制可直接通过针对HSV - 2的抗病毒的治疗就可以得到抑制。
He said that the paper written by Dr Nagot and colleagues has "direct clinical implications, suggesting that HIV-1 replication can be reduced with antiviral therapy directed solely at HSV-2."
迄今为止，全称异嗜性小鼠白血病病毒相关病毒(Xenotropicmurine leukemia virus - related virus)的XMRV病毒的复制速度似乎不像艾滋病毒那么快。
So far, XMRV, known fully as xenotropic murine leukemia virus-related virus, doesn't appear to replicate as quickly as HIV does.
许多研究报告说，疱疹病毒复制起点附近存在异常的反向重复。
A number of studies reported there are unusual inverted repeats in the neighborhood of the replication origins of herpesviruses.
与细菌不同，病毒包括没有复制所需的所有机制，因此它们要像寄生虫一样向其它细胞借产品。
Unlike bacteria, viruses don't contain all the machinery necessary to replicate, and so like parasites they borrow the goods from other cells.
他们报告说，该病毒的快速复制表明它适应了人类细胞的环境。
The virus's rapid replication indicates that it is adapted to human cells, they reported.
病毒基因组分为八段，它会复制并组装每一段基因，使其成为新的病毒。
The virus’ genome has eight segments, each of which it copies and assembles into new viruses.
这种感染不会损伤细胞，但是当病毒复制并感染新的癌细胞时，发光的蛋白质也会增多，从而使肿瘤变得越来越亮。
The infection did not harm the cells, but as the virus replicated and infected new cancer cells, the glowing protein multiplied too. This caused the tumours to grow brighter and brighter.
这种感染不会损伤细胞，但是当病毒复制并感染新的癌细胞时，发光的蛋白质也会增多，从而使肿瘤变得越来越亮。
The infection did not harm the cells, but as the virus replicated and infected new cancer cells, the glowing protein multiplied too.This caused the tumours to grow brighter and brighter.
这种感染不会损伤细胞，但是当病毒复制并感染新的癌细胞时，发光的蛋白质也会增多，从而使肿瘤变得越来越亮。
The infection did not harm the cells, but as the virus replicated and infected new cancer cells, the glowing protein multiplied too.This caused the tumours to grow brighter and brighter.
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