为了应对DNA损伤，细胞有一套复杂的机制，包括细胞周期停滞，诱导转录和DNA修复被激活。
In response to DNA damage, the complex cellular mechanisms including cell cycle arrest, transcription induction and DNA repair are activated.
建立于目标基因激活和失活基础之上的几个化学分子诱导基因表达系统已有报道。
Various chemical inducible systems based on activation and inactivation of the target gene had been described.
牙周优势菌lps作用于单核巨噬细胞，诱导产生各种细胞因子和炎性介质，参与牙周病的炎症激活和组织创伤。
LPS involves in the inflammatory process and tissue destruction during periodontal diseases through inducing mononuclear macrophages to produce various cell factors and inflammatory mediators.
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