该综述将对FAK与肿瘤的关系以及FAK作为肿瘤治疗靶点的研究进展进行探讨。
This review is to summarize the relationship between FAK and tumor progression, and to discuss the strategies targeting FAK for cancer treatment.
一种治疗药物以突变的基因为靶点，该基因突变后能让肿瘤细胞持续增多。另一种药物能激活病患免疫系统对抗疾病。
One drug specifically targets a mutated gene that tells a cancer cell to grow, the other boosts a patient's immune system in the fight against the disease.
另外，在非肿瘤治疗领域，随着从分子生物学角度更好的理解疾病，对于特定靶点药物的研究有所增加。
Also, in nononcologic therapeutic areas understanding the molecular biology of the disease increases; thus, the chances of developing specific targeted drugs increase.
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