治疗组肝细胞脂肪变性程度无显著变化。
And the degree of hepatocyte steatosis did not change in the treatment group.
模型组大鼠肝小叶结构紊乱，有大量纤维组织沉积，炎症细胞浸润，肝细胞脂肪变性。
Lobules of liver in the rats of model group was disorder with a pile of deposition of fibrous tissue inflammatory cell infiltration and fatty degeneration of hepatocytes.
目的以人正常肝细胞株L-02为实验材料，采用油酸诱导建立培养肝细胞脂肪变性模型。
Objective To explore the effects of selenium on DNA damage and apoptosis of L-02 cell induced by fluoride.
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