治疗组肝细胞脂肪变性程度无显著变化。
And the degree of hepatocyte steatosis did not change in the treatment group.
模型组大鼠肝小叶结构紊乱，有大量纤维组织沉积，炎症细胞浸润，肝细胞脂肪变性。
Lobules of liver in the rats of model group was disorder with a pile of deposition of fibrous tissue inflammatory cell infiltration and fatty degeneration of hepatocytes.
目的以人正常肝细胞株L-02为实验材料，采用油酸诱导建立培养肝细胞脂肪变性模型。
Objective To explore the effects of selenium on DNA damage and apoptosis of L-02 cell induced by fluoride.
结论中药脂肝宁通过抑制氧化应激、稳定肝细胞膜、抑制脂质过氧化来改善酒精所致肝细胞脂肪变性。
The steatosis state of liver was improved. Conclusion ZGN improves alcoholic fatty liver by suppressing oxidative stress, stabilizing the hepatic cellular membrane and inhabiting lipid peroxidation.
脂肪性肝病是多种原因引起的病变主体在肝小叶、以肝细胞脂肪变性和脂肪蓄积过多为主的临床病理综合征。
Fatty liver disease (FLD) is a clinicopathological syndrome caused by a variety of reasons. The main lesions are in hepatic lobule with steatohepatitis and accumulation of triglycerides in the liver.
伴中度脂肪变性的小结节型肝硬化。注意再生的肝细胞小结节被汇管区之间的纤维结缔组织包围。
Micronodular cirrhosis is seen along with moderate fatty change. Note the regenerative nodule surrounded by fibrous connective tissue extending between portal regions.
高剂量组小鼠可见肝细胞轻度脂肪变性。
Light fatty degeneration was found in liver cells of mice in higher dose group.
病理观察显示，番茄红素各剂量组脂肪变性及肝细胞肿胀程度减轻，接近正常水平。
Pathological observation showed that fatty degeneration and liver cell swelling were relieved in all the dosage groups, and were close to the normal level.
结果发现，肝脏脂肪变性与急性严重感染有密切关系，其脂肪变性主要与感染、毒素等损害了肝细胞内线粒体及其酶的活性有关。
The results showed that liver steatosis was closely related to the serious acute infection, in which the infection or toxin might damage enzyme and thread granules in the liver cells.
结论实验性大鼠脂肪肝肝细胞发生脂肪变性，肝脂、血脂含量同时升高，并有肝细胞损伤。
Conclusion in the experiments, the high lipid food rats displayed the liver cells fat denaturalization, the blood lipid and liver lipid increased with liver cells damage.
方法：取正常人肝l - 02细胞，和经50%胎牛血清造模24小时建立的脂肪变性肝细胞模型，共同作为研究对象。
Methods: Use L-02 hepatocyte of normal human and the hepatocyte under fatty change after exposure to 50% fetal bovine serum for 24 hours as study objects.
方法：取正常人肝l - 02细胞，和经50%胎牛血清造模24小时建立的脂肪变性肝细胞模型，共同作为研究对象。
Methods: Use L-02 hepatocyte of normal human and the hepatocyte under fatty change after exposure to 50% fetal bovine serum for 24 hours as study objects.
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