fibrillary contraction
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毛细血管损伤促进了肌原纤维收缩带形成，加速了心肌细胞坏死。
Injuries of capillaries promoted the formation of contraction band (CB) of myofibril and myocardial necrosis.
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在变圆过程中微管及肌原纤维解聚，自发性收缩停止。
During the rounding process spontaneous contractions stopped and microtubules and myofibrils depolymerized.
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