毛细血管损伤促进了肌原纤维收缩带形成，加速了心肌细胞坏死。
Injuries of capillaries promoted the formation of contraction band (CB) of myofibril and myocardial necrosis.
在变圆过程中微管及肌原纤维解聚，自发性收缩停止。
During the rounding process spontaneous contractions stopped and microtubules and myofibrils depolymerized.
在变圆过程中微管及肌原纤维解聚，自发性收缩停止。
During the rounding process spontaneous contractions stopped and microtubules and myofibrils depolymerized.
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