这些数据表明晶状体蛋白的非酶糖基化可能会导致白内障。
These data suggest that nonenzymatic glycosylation of lens crystallins may contribute to cataract formation.
品状体结构蛋白的非酶糖基化引起晶状体构象变化在糖尿病性白内障的发生中起重要作用。
It is vital in the diabetic cataract formation that the non-enzymic glycosylation of the lens structural proteins by glucosamine induces conformational changes in the lens.
观察了香烟烟雾对大鼠晶状体的脂质过氧化作用以及对抗氧化酶活性和非蛋白质巯基水平的影响。
The effects of cigarette smoking on lipid peroxidation, the activities of antioxidases, and the level of non protein sulphydryl group in the rat lenses were investigated.
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