这些数据表明晶状体蛋白的非酶糖基化可能会导致白内障。
These data suggest that nonenzymatic glycosylation of lens crystallins may contribute to cataract formation.
品状体结构蛋白的非酶糖基化引起晶状体构象变化在糖尿病性白内障的发生中起重要作用。
It is vital in the diabetic cataract formation that the non-enzymic glycosylation of the lens structural proteins by glucosamine induces conformational changes in the lens.
观察了香烟烟雾对大鼠晶状体的脂质过氧化作用以及对抗氧化酶活性和非蛋白质巯基水平的影响。
The effects of cigarette smoking on lipid peroxidation, the activities of antioxidases, and the level of non protein sulphydryl group in the rat lenses were investigated.
目的观察香烟烟雾对大鼠晶状体的脂质过氧化作用以及对抗氧化酶活性和非蛋白质巯基的影响。
ObjectiveTo observe the effect of cigarette smoke on the lipid peroxidation, the activity of antioxidases, and the level of non protein sulphydryl group in the rat lenses.
基质金属蛋白酶-2,9及其抑制因子- 1,2在老年性白内障晶状体上皮细胞的表达。
Expression of Matrix Metalloproteinase-2, -9 and Their Inhibitors TIMP-1, -2 on Lens Epithelial Cells of Aged-related Cataract.
中药制剂的实验药理研究主要集中在抗氧化作用、抑制醛糖还原酶作用和抑制晶状体上皮细胞凋亡方面。
Pharmacological experiments of Chinese herbal medicine were focused on antioxidant effect and inhibiting aldose reductase and decreasing apoptosis of lens epithelial cell.
两者协同作用能有效地修复晶状体蛋白质和酶的巯基，维持晶状体的功能。
The two enzymes may work synergistically to repair thiols in lens proteins and enzymes and maintain the function of the lens effectively.
目的探讨在紫外线诱导的人晶状体上皮细胞DNA损伤修复过程中端粒酶活性和氧化损伤蛋白的变化和作用。
Objective To investigate the changes and the role of telomerase activity and other stress-related proteins in the process of UV-induced DNA damage and repair in human lens epithelial cells.
结果0 .2 5 %、0 .2 %胰蛋白酶溶液处理的晶状体前囊，晶状体上皮细胞可以完全脱落。
Results 0.25%? 0.2% trypsin can clean up lens epithelial cells from anterior capsules totally.
结果0 .2 5 %、0 .2 %胰蛋白酶溶液处理的晶状体前囊，晶状体上皮细胞可以完全脱落。
Results 0.25%? 0.2% trypsin can clean up lens epithelial cells from anterior capsules totally.
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