结果发现，IPF主要表现为剧烈干咳、进行性气短、紫绀、杵状指趾和肺部捻发音。

The results showed that the main manifestations of IPF were severe dry cough, progressive short breath, cyanosis, clubbed fingers and crepitant rales in lungs.

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对控制多趾的相关基因及其分子机制进行研究，有利于我们深入了解多指（趾）畸形的形成机制，揭示肢体发育的过程和机理。

Study the genes of polydactyly and molecular mechanism can help us to know how limbs form and reveal the producing process of limbs.

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本病的基础是因正常血管反应的局部缺陷而导致的指(趾)动脉和皮肤小动脉的异常血管收缩。

Abnormal vasoconstriction of digital arteries and cutaneous arterioles due to a local defect in normal vascular responses is thought to underlie the disorder.

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