严重休克病人可出现进行性缺氧和呼吸困难,造成低氧血症性呼吸衰竭,称为休克肺。(或称急性呼吸窘迫综合征 ARDS)。休克肺是休克患者死亡的重要原因之一。
结论:肠淋巴管结扎干预休克肺损伤的机制与上调或下调上述相关基因的表达有关。
CONCLUSION: the results indicate that the mechanism of mesenteric lymph ligation alleviating the lung injury after shock is related to up or down regulation of the above genes.
虎杖甙对内毒素休克大鼠肺组织中磷脂酶a2的影响。
Effect of polydatin on phospholipase A2 in lung tissues in rats with endotoxic shock.
在白细胞减少并RES封闭后失血性休克过程中,发现血小板聚集度降低,肺损伤不发生。
It was found that the degree of platelets aggregation was lower and pulmonary injury did not occur during leukopenia with hemorrhagic shock after blockade of RES.
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