结论：肠淋巴管结扎干预休克肺损伤的机制与上调或下调上述相关基因的表达有关。
CONCLUSION: the results indicate that the mechanism of mesenteric lymph ligation alleviating the lung injury after shock is related to up or down regulation of the above genes.
虎杖甙对内毒素休克大鼠肺组织中磷脂酶a2的影响。
Effect of polydatin on phospholipase A2 in lung tissues in rats with endotoxic shock.
方法建立豚鼠异种血清过敏性休克模型，采用专性底物对模型鼠的血清，肺，气管类胰蛋白酶活力进行测定。
Methods Sera and tissues were collected from anaphylactic shock guinea-pigs, and the enzyme activity was tested colormetrically using special substrate, BAPNA.
在白细胞减少并RES封闭后失血性休克过程中，发现血小板聚集度降低，肺损伤不发生。
It was found that the degree of platelets aggregation was lower and pulmonary injury did not occur during leukopenia with hemorrhagic shock after blockade of RES.
结果表明白细胞和血小板参与RES抑制后失血性休克引起的肺损伤变化。
The results suggested that leukocytes and platelets involved in the pulmonary injury of acute hemorrhagic shock after depressed RES.
目的：研究葛根素对实验性失血性休克家兔导致急性肺损伤(ALI)的保护作用。
AIM: To study the protective effects of puerarin on acute lung injury (ALI) which was a result of experimental hemorrhagic shock in rabbits.
结果在失血性休克和LPS刺激后肺出现明显的中性粒细胞浸润、红细胞渗出。
Results Hemorrhagic shock and LPS challenge increased lung neutrophil infiltration and erythrocyte diapedesis.
目的评价血管外肺水指数（EVLWI）对感染性休克患者预后判断的意义。
ObjectiveTo investigate the prognostic value of extravascular lung water index (EVLWI) in critically ill patients with septic shock in intensive care unit (ICU).
目的：探讨在热休克条件下，人胚肺二倍体细胞MRC-5株诱导型热休克蛋白70（HSP70）在细胞水平的表达机制，以及对MRC-5细胞衰老的影响。
Objective: To explore the expressive mechanism of induced HSP70 and it' s effect on the senescence of MRC-5 cell in the heat shock condition.
结论：丙酮酸乙酯可以对抗内毒素诱导的脓毒症休克犬的急性肺损伤。
CONCLUSION: Ethyl pyruvate has protective effects on acute lung injury in dogs with septic shock.
方法：观察双黄连注射液对内毒素休克小鼠的生存时间及对内毒素所致大鼠肝、肺、肾等病理改变的影响。
METHODS To observe the effect of Shuanghuanglian injection on the average death time of mice and the pathological changes of rats livers , lungs and kidneys induced by endotoxin.
目的分析临床常用液体对创伤失血性休克液体复苏后肺组织的影响，为创伤失血性休克实施理想的液体复苏治疗提供依据。
Objective to analyse the effects of different resuscitative fluids on the lungs following hemorrhage, and to provide the scientific basis for the perfect fluid treatment in hemorrhage.
目的：采用大鼠肠缺血再灌注模型，对创伤休克后多系统器官衰竭中肺损伤的发生机制进行研讨。
Objecctive: To explore the pathogenesis of lung injury during multiple system organ failure (MSOF) after traumatic shock by using intestinal ischemia reperfusion model of the rat.
目的：采用大鼠肠缺血再灌注模型，对创伤休克后多系统器官衰竭中肺损伤的发生机制进行研讨。
Objecctive: To explore the pathogenesis of lung injury during multiple system organ failure (MSOF) after traumatic shock by using intestinal ischemia reperfusion model of the rat.
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