Pathological scars may be divided into two kinds: hypertrophic scars and keloids.

病理性瘢痕在临床上可分为增生性瘢痕和瘢痕疙瘩两种类型。

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Objective To investigate the differences of the gap junctional communication among fibroblasts derived from pathological scars and normal skin.

目的研究和比较不同来源的成纤维细胞缝隙连接介导的细胞间通讯的差异。

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Conclusion SP may play an important role in the formation of pathological scars by modulating the expression of apoptosis-associated genes, which is mediated by SP receptor.

结论SP通过调控成纤维细胞凋亡相关基因的表达参与病理性瘢痕形成，该作用由SP受体介导。

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