Pathological scars may be divided into two kinds: hypertrophic scars and keloids.
病理性瘢痕在临床上可分为增生性瘢痕和瘢痕疙瘩两种类型。
The above-mentioned results provide technological methods and theoretical basis for gene therapies for pathological scars.
本研究为病理性癫痕的临床基因治疗研究奠定了一定的理论基础，并提供了一定的技术手段。
Objective To investigate the differences of the gap junctional communication among fibroblasts derived from pathological scars and normal skin.
目的研究和比较不同来源的成纤维细胞缝隙连接介导的细胞间通讯的差异。
Conclusion SP may play an important role in the formation of pathological scars by modulating the expression of apoptosis-associated genes, which is mediated by SP receptor.
结论SP通过调控成纤维细胞凋亡相关基因的表达参与病理性瘢痕形成，该作用由SP受体介导。
Conclusion SP may play an important role in the formation of pathological scars by modulating the expression of apoptosis-associated genes, which is mediated by SP receptor.
结论SP通过调控成纤维细胞凋亡相关基因的表达参与病理性瘢痕形成，该作用由SP受体介导。
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