结果：高血压病人存在内皮依赖性的血管舒张功能障碍，血管内皮合成或释放一氧化氮明显减少，引起高血压。

Resu LTS: Patients with essential hypertension have abnormal endothelium-dependent vascular relaxation. Reduction of nitric oxide synthesized and released by vascular endothelium causes hypertension.

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SEB对非特异性免疫系统也具有类似的作用，表现在巨噬细胞释放一氧化氮量增加及胞内酸性磷酸酶活性增强。

Similarly, SEB can increase the amount of NO and the activation of ACP in the non-specific immune responses.

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内皮细胞可以释放一种保护我们血管的神奇气体分子——一氧化氮。

Endothelial cells manufacture a magical protective molecule of gas called nitric oxide, which protects our blood vessels.

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