结果:高血压病人存在内皮依赖性的血管舒张功能障碍,血管内皮合成或释放一氧化氮明显减少,引起高血压。
Resu LTS: Patients with essential hypertension have abnormal endothelium-dependent vascular relaxation. Reduction of nitric oxide synthesized and released by vascular endothelium causes hypertension.
SEB对非特异性免疫系统也具有类似的作用,表现在巨噬细胞释放一氧化氮量增加及胞内酸性磷酸酶活性增强。
Similarly, SEB can increase the amount of NO and the activation of ACP in the non-specific immune responses.
内皮细胞可以释放一种保护我们血管的神奇气体分子——一氧化氮。
Endothelial cells manufacture a magical protective molecule of gas called nitric oxide, which protects our blood vessels.
血管内皮功能紊乱主要原因是血管内皮一氧化氮(NO)的释放量减少而失去了NO的血管保护作用。
The main reason of dysfunction of vascular endothelium is the decreasing NO release of vascular endothelium which leads to lose protect function of NO.
目的:研究l -精氨酸对家兔内源性一氧化氮释放和血液流变性的影响。
Objective: To study the effects of L-arginine on endogenous nitric oxide release and hemorheology in rabbits.
一氧化氮是植物合成和释放的一种气体,也可作为信号分子调节植物的生长和发育。
Nitric oxide is a gaseous molecule that synthesized and released by plants, it control growth and development of plants as a signal molecule.
结论脊髓伤后脊髓组织中一氧化氮(no)与eaa的释放相互促进,因而在继发性脊髓损伤中形成级联放大的神经毒性因子释放。
Conclusion no and EAA can promote each other, and result in cascade release of nervous noxious factors in secondary spinal cord injury.
结论一氧化氮可减少肝缺血再灌注损伤后肝酶的释放,抑制肝细胞的凋亡,改善肝缺血再灌注损伤。
Conclusion Nitric oxide could increase the release of the hepatic enzymes after liver ischemia reperfusion injury, inhibit the apoptosis of hepatocytes and improve liver ischemia reperfusion injury.
动物的排泄物释放出的气体,例如氢化硫、氨气、污染了农场周围的空气,同时,甲烷和一氧化氮则是导致全球变暖的主要元凶。
Animal excrement emits gases, such as hydrogen sulfide and ammonia, that poison the air around farms, as well as methane and nitrous oxide, which are major contributors to global warming.
在不考虑其他因素的情况下,内皮释放的一氧化氮增加或减少会同时引起血压的下降或升高。
Without considering other factors, the ascending or descending of NO derived from endothelium will increase or decrease the blood pressure.
我们也关注红树林湿地一氧化氮的释放规律和一氧化氮的生理作用及其作用机理研究。
We also focus on the nitric oxide emission from mangrove wetland and the physiological roles of nitric oxide in plant development.
目的研发一种通过增加一氧化氮(NO)释放从而选择性改善脉络膜血流的新型药物,用于治疗年龄相关性黄斑变性(amd)。
OBJECTIVE To synthesize a compound, which can specifically facilitate choroidal blood flow via increasing of nitric oxide (NO), for the treatment of age-related macular degeneration (AMD).
目的:探讨脑缺血时微血管管径与一氧化氮释放的关系。
OBJECTIVE: To investigate the correlation between the microvascular diameter and NO in cerebral ischemia.
目的:探讨药物静脉注射致死对大鼠肾脏内皮素和一氧化氮释放的影响。
Objective To study the effect on renal endothelium and nitric oxide synthetase in rats by lethal drug intravenous administration.
结果表明硝苯地平能够有效抑制上述细胞一氧化氮的释放,且抑制效应呈剂量依赖关系。
The results demonstrated that nifedipine can inhibit the production of nitric oxide in T629 cells, CPAE cells and SM3 cells, and also inhibit the amount of NO stimulated by LPS in macrophages.
不同于以往的一氧化氮的产品,本品不仅能促进一氧化氮的生成,更兼顾促进一氧化氮的有效释放。
Differing from previous nitric oxide products, this product not only stimulates generation of nitric oxide, but also promotes its effective release.
结论大鼠慢性缺氧时一氧化氮释放减少,而一氧化氮释放减少可能参与缺氧性肺动脉高压的形成。
Conclusion Nitric oxide release decreased during chronic hypoxia, which was probably involved in the mechanism of hypoxic pulmonary hypertension.
结论大鼠慢性缺氧时一氧化氮释放减少,而一氧化氮释放减少可能参与缺氧性肺动脉高压的形成。
Conclusion Nitric oxide release decreased during chronic hypoxia, which was probably involved in the mechanism of hypoxic pulmonary hypertension.
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