可见，肿瘤细胞上共刺激分子缺陷是肿瘤免疫逃逸的重要机制之一。

Therefore, deficiency of costimulatory molecules on tumor cells is one of the important mechanisms of tumor immune escape.

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近年来，肿瘤坏死因子受体超家族成员- OX40 (CD134)，作为一共刺激分子对于维持CD 4 + T细胞的活化、增值、存活起到关键性作用。

In recent years, tumor necrosis factor receptor (TNFR) family member-OX40 (CD134), as a key co-stimulatory molecule, involved in maintenance of activation, proliferation, survival of CD4 + t cells.

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目的：研究银屑病患者外周血T淋巴细胞表面共刺激分子CD80的表达及其与银屑病皮损面积和严重程度指数（PASI）的相关性。

Objective: To measure the expression of co-stimulatory molecule CD80 on peripheral blood T lymphocytes in patients with psoriasis and its correlation with psoriasis area and severity index (PASI).

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