结果：粒、红、巨核三系均有不同程度的病态造血表现。
The results showed that there were abnormal hematopoiesis in granulocyte series, erythron series and megakaryocytic series in different extent.
结论巨核祖细胞的内在缺陷是APATP的主要发病机制。
Conclusion The intrinsic defect of megakaryocyte progenitor cell is considered to be a primary pathogenesis of APATP.
目的：探讨造血刺激因子对巨核系成熟细胞体外扩增的影响。
Objective To investigate the effect of hematopoietic stimulating factors on the expansion of mature megakaryocytes.
目的：探讨骨髓成纤维细胞条件培养液对体外巨核系细胞生成的促进作用。
Objective To investigate the effect of bone marrow fibroblasts conditioned media on the expansion of megakaryocytes in vitro.
结果显示，ABCG2的表达促进了PMA诱导的K562向巨核系的分化。
Results showed that ABCG2 expression increased megakaryocytic differentiation of K562 cells induced by PMA.
结论 外周血网织血小板数量可反映骨髓巨核系统生成血小板的情况，从而用于疾病的诊断和鉴别诊断。
Conclusion IPF in peripheral blood can reflect the amount of platelets produced by megakaryocytes, thus contributing to the diagnosis and differential diagnosis of hematological diseases.
前言： 目的：探讨人微小病毒B19（H PVB19）感染的特发性血小板减少性紫癜（ITP）患儿血清对人巨核祖细胞（CFU-MK）增殖的影响。
Objective:To investigate the effects of serum infected by human parvovirus B19(HPVB19) on the proliferation of colony forming unit-megakaryocyte (CFU-MK) in ITP patients.
恶性贫血，有巨卵形红细胞、多叶核嗜中性粒细胞。
This hypersegmented neutrophil is present along with macro-ovalocytes in a case of pernicious anemia.
本文讨论了高激发核中的同位旋标量表面巨共振和偶极巨共振。
The isoscalar giant surface resonance and giant dipole resonance in highly excited nuclei are discussed.
本文考察了核物理研究的新进展，从规则运动与无规运动并存与交织的观点，评述了原子核巨共振的研究；
The study of nuclear giant resonances has been reviewed from the viewpoint of interplay between nuclear regular and irregular motions.
在原于核巨共振方面介绍了矮共振区的GDR研究，形变核的GDR以及高温转动核的GDR研究。
Brief introductions in the field of GDR studies of pygmy resonance and GDR studies in deformed...
在原于核巨共振方面介绍了矮共振区的GDR研究，形变核的GDR以及高温转动核的GDR研究。
Brief introductions in the field of GDR studies of pygmy resonance and GDR studies in deformed...
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