Nature magazine published online a research paper on Wednesday, claiming that virus-specific cells, CD8 + t, are able to control viral replication in both animal models and HIV infections.
星期三，《自然》杂志发表了一篇研究成果，指出CD 8 +T细胞亚类能抑制动物模型和艾滋病感染的病毒复制。
Preventing the virus from replicating can help the immune system recover from the HIV infection and improve T-cell counts.
抑制病毒的复制，可以使免疫系统在受到感染后进行自我修复，也可以增加T细胞数量。
One of the causes of viral dormancy is that the T-cell the virus resides in is itself in a resting state.
病毒产生休眠的原因之一就是病毒寄居的T细胞本身就是休眠状态。
The researchers engineered a virus that infected a patient's T-cells, a type of white blood cell, and carried instructions to bind to cancer cells and ultimately kill them.
研究人员研制出一种病毒，这种病毒可以感染患者的一种白血细胞T型细胞，并接受指令与癌细胞结合在一起，并最终杀死癌细胞。
"Minocycline reduces the capability of the virus to emerge from resting infected t cells," Szeto explains.
“二甲胺四环素降低了病毒脱离休眠状态的感染T细胞的能力，” Szeto解释说。
In the lab, they infected those cells with a virus carrying genes that create T-cell receptors, essentially homing devices for, in this case, melanoma.
在实验室，他们用病毒感染那些细胞，这种病毒携带有能产生T细胞受体的基因，在这个例子中，实质上就是产生黑色素瘤导归器的基因。
The model also showed that once those T cells are released into the bloodstream, they can effectively attack HIV proteins, even when the virus mutates.
这个模型显示，一旦T细胞释放入血，它们就可以强烈攻击HIV蛋白，即使它们已经变异了。
Unlike the drugs used in HAART which target the virus, minocycline homes in on, and adjusts t cells, major immune system agents and targets of HIV infection.
HAART以病毒本身为靶子，与这种疗法使用的药物有所不同的是，二甲胺四环素采取的是导向目标追踪方式，调节免疫系统的主要行动者以及HIV感染目标T细胞。
But even with the drugs, the virus stays in the body's "T-cells," which are T-lymphocytes.
但是即使服用了这些药物，病毒仍停留在人体的T细胞，也就是T淋巴细胞之中。
Instead, the virus required cell activation for replication and mediated cell fusion of t lymphocytes.
相反，该病毒需要细胞激活介导的复制和细胞融合的T淋巴细胞。
This level of replication sufficient to infection near the T-lymphocytes, so that a period of dormancy memory t cells in virus hidden library has been expanded.
这样的复制水平足以感染临近的T淋巴细胞，使休眠期记忆性T细胞中的病毒潜藏库不断得到扩充。
Objective:To detect the clinicopathologic features of nasal NK/T-cell lymphoma in Guangxi and their association with Epstein-Barr virus (EBV).
目的：研究广西地区鼻NK/T细胞淋巴瘤的临床病理特点及与EB病毒（EBV）的关系。
Methods RT PCR products were amplified from S85 46 virus infected cells, cloned into T vector, sequenced and analyzed using DNASTAR software.
方法将特异性引物从S8546病毒感染细胞PCR扩增的产物克隆于T载体，正确的克隆纯化后测序，应用DNASTAR软件比较分析。
Experiment proved that minocycline could greatly reduce the virus load, but also affects T-cell activation and reproduction.
试验证明，二甲胺四环素可极大地降低病毒量，而且影响T细胞的活化与繁殖。
Eventually, the piggybacked antibody also tags the invading virus for destruction by other immune cells, known as t cells.
最终，黏附着的抗体为其他被称为T细胞的免疫细胞将侵入的病毒标记起来，让其破坏。
In contrast, virus persistence is generally associated with impairment, especially of t cell responses.
相反，病毒持续存在则一般与免疫应答尤其是T细胞应答受损有关。
The virus particles attack T-lymphocytes, stealing their genetic machinery, thereby forcing them to produce more copies of the virus.
该病毒粒子攻击T淋巴细胞，盗窃它们的遗传基因机制，从而迫使它们录制更多的病毒复制数目。
Influenza virus cannot directly induce apoptosis of PHA-stimulated t lymphocytes.
流感病毒不能直接诱导PHA刺激的T淋巴细胞凋亡。
The cytotoxic t cells kill the RV infected cells to achieve virus clearance.
细胞毒性T细胞则通过杀伤rv感染的细胞以清除病毒感染。
Objective To study the roles of dengue (den) virus specific t cells in the pathogenesis of den virus infection.
目的研究登革病毒特异性T细胞在登革病毒致病中的作用。
The drug and virus reactivation destroy tissues by an immune allergic reaction. And the destruction is primarily caused by CD8 + cytotoxic t lymphocytes.
药物及病毒再激活引发的免疫过敏反应所致组织损害主要南cd 8 +细胞毒性T淋巴细胞造成。
We found a direct correlation between the level of competition among t cells and the rate at which the virus escaped.
我们发现了T细胞竞争的水平和逃避免疫的病毒相对数量之间的直接联系。
AIDS (AIDS) by the human immunodeficiency virus (HIV) infection caused by defects in t cell immune function mainly of a mixed immune deficiency disease.
艾滋病(AIDS)是由人类免疫缺陷病毒(HIV)感染引起的以T细胞免疫功能缺陷为主的一种混合免疫缺陷病。
What is even more exciting is that the spread of the virus spreading at the same time show a certain rate of decrease, and the loss of ITK feature does not affect the normal T-cell survival.
更令人兴奋的是，病毒的传播蔓延同时呈现一定幅度减小，且损失itk功能后并不影响T细胞的正常存活。
Studies performed in the HCV infected animal model of virus infection showed initially that by blocking this pathway it is possible to reconstitute the T cell function.
在HCV感染的动物模型中所进行的研究显示，阻断这条途径就有可能重建T细胞功能。
Objective To obtain the prevalence data on human T Lymphotropic virus (HTLV) infection in 2 339 blood donors of an endemic coastal region of Fujian, China.
目的了解人类T淋巴细胞白血病病毒（HTLV）流行区献血员中HTLV感染情况。
Researchers have realized that the good effect in the treatment, the virus was not detected in blood, some T-cells and other cells appears to have a very low level in order to generate a new virus.
研究人员也意识到，在治疗效果很好、血液中检测不到病毒的情况下，一些辅助性T细胞和其他细胞似乎也在以极低的水平产生新的病毒。
The third phase, AIDS, occurs when the virus has changed so much that the body's t cells are no longer effective at keeping it in check.
在第三阶段，AIDS病毒已发生巨大改变，人体的T细胞便不再能有效的检测到了。
The third phase, AIDS, occurs when the virus has changed so much that the body's t cells are no longer effective at keeping it in check.
在第三阶段，AIDS病毒已发生巨大改变，人体的T细胞便不再能有效的检测到了。
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