Ouabain was added in order to induce oscillatory afterpotentials.
哇巴因诱发振荡后电位。
Objective To study the relationship between the concentration of serum endogenous ouabain (EO) and glomerular diseases.
目的探讨内源性哇巴因（EO）与肾小球疾病的关系。
Methods:Anti-ouabain polyclonal antibody egg yolk(IgY) and anti-ouabain rabbit antibody(IgG) were prepared respectively.
方法：分别制备出鸡蛋黄抗哇巴因多克隆抗体及兔抗体。
However, the effect of Berberine to prolong lethal time induced by Ouabain is less effective than Sodium Phenytoin, Quinidine and Propranolol.
但黄连素延长哇巴因对豚鼠的致死时间作用不及苯英钠、奎尼丁及心得安等药物。
The results indicated that petroleum ether extract had the ability to antagonize toxicity induced by ouabain, while water extract had little effect on it.
未见水提物抗哇巴因所致豚鼠心脏毒性及抗氧自由基的作用。
Furthermore, XT not only attenuated the positive inotropic action, but also delayed the following toxicity response induced by ouabain in the isolated left atria.
XT对哇巴因的正性肌力作用及随后出现的心肌收缩力降低的毒性反应均有明显的对抗作用。
Objective: To study the antiarrhythmic activity of taurine-magnesium coordination compound (TMC) in vivo by using the arrhythmic model induced by ouabain of guinea pig.
目的：建立哇巴因诱发的豚鼠心律失常模型，观察新型配合物牛磺酸镁（TMC）的在体抗心律失常作用；
Bioactivity test on isolated rat heart failure model suggested 1 could increase coronary blood flow and myocardial con - tractive power compared with positive control ouabain.
离体大鼠心衰试验表明，与对照药哇巴因比较，化合物1能增加冠脉血流量和增强心肌收缩力。
Conclusion THP had the effect of decreasing the amplitude of DAD of rabbit heart induced by ouabain thus reducing the chance of triggered ventricular arrhythmia induced by DAD.
结论四氢巴马汀具有降低哇巴因诱发家兔在体心脏延迟后除极振幅的作用，从而减少由延迟后除极所引起的触发性室性心律失常的发生。
Myofibrillar fragmentation, swelling of the cardiac myocytes, absence of the Z line, increases of the mitochondria and collagen fibers were found in ouabain group by transmission electron microscopy.
透射电镜观察显示，哇巴因组大鼠心肌细胞肿胀，肌纤维断裂，Z线消失，线粒体增生肿胀，部分空泡化，细胞间胶原纤维增生。
Myofibrillar fragmentation, swelling of the cardiac myocytes, absence of the Z line, increases of the mitochondria and collagen fibers were found in ouabain group by transmission electron microscopy.
透射电镜观察显示，哇巴因组大鼠心肌细胞肿胀，肌纤维断裂，Z线消失，线粒体增生肿胀，部分空泡化，细胞间胶原纤维增生。
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