Methods Human umbilical vein endothelial cells (HUVECs) were cultured in vitro.
方法体外培养人脐静脉内皮细胞，通过传代形成细胞的衰老模型。
The changes in cell cycle progression of synchronized HUVECs were examined by flow cytometric analysis.
用流式细胞技术检测各组细胞周期，观察细胞增殖能力的变化。
The cultured HUVECs were divided into 6 groups according to different experimental agents: control ( serum - free DMEM ) ;
施加实验因素，分成6组：正常对照组（无血清DMEM）；
Objective To find the morphological proof of direct communication between human umbilical vascular endothelial cells (HUVECs).
目的寻找人脐静脉内皮细胞之间的直接通讯的形态学证据。
CONCLUSION: APC can inhibit HUVECs apoptosis induced by LPS and promote cell proliferation, thus protect the cells from injury.
结论：APC拮抗lps诱导的血管内皮细胞凋亡并促进细胞的增殖，发挥保护细胞的作用。
Results: In cellular experiment, Ginseng and notoginseng can promoting the HUVECs to proliferates secrete and express VEGF markedly.
结果：细胞实验，结果表明三七和人参果提取物能显著促进HUVEC增殖、分泌和表达VEGF等。
Objective: To research the effects on proliferation of human umbilical vein endothelial cells (HUVECs) induced by different doses of iodine.
目的：探讨不同碘浓度对人脐静脉血管内皮细胞增殖的影响。
The effect of the recombinant protein on proliferation of human umbilical vein endothelial cells (HUVECs) was also analyzed using the MTT assay.
采用噻唑蓝（MTT）比色法测定表达蛋白抑制血管内皮细胞的活性。
Objective To study the effects of VEGF secreted by hypoxia-cultured cardiomyocytes on proliferation and migration of cultured human umbilical vein endothelial cells (HUVECs).
目的研究低氧培养心肌细胞分泌出的血管内皮生长因子(VEGF)蛋白对培养内皮细胞增殖和移行的影响。
Objective To investigate the effect of telmisartan on the protein and gene expression of angiotensin-converting enzyme 2 (ACE2) in human umbilical vein endothelial cells (HUVECs).
目的探讨替米沙坦对人血管内皮细胞血管紧张素转换酶2（ACE2）表达的影响。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
Conclusion the mechanism of puerarin reducing the overload of calcium in HUVECs may be related with blocking both voltage-dependent calcium channels and the release of calcium pool in endoplasm.
结论葛根素可以缓解培养人脐静脉内皮细胞内钙超载，其作用机制可能与拮抗膜上电压依赖型钙通道和抑制内质网钙库释放有关。
Conclusion the mechanism of puerarin reducing the overload of calcium in HUVECs may be related with blocking both voltage-dependent calcium channels and the release of calcium pool in endoplasm.
结论葛根素可以缓解培养人脐静脉内皮细胞内钙超载，其作用机制可能与拮抗膜上电压依赖型钙通道和抑制内质网钙库释放有关。
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