Results: SI of CADASIL group was significantly higher than that of control group in both GM and WM.
结果：CADASIL组白质及灰质小动脉的硬化指数明显高于对照组。
Conclusions: in CADASIL long penetrating arterioles or branches supplying subcortical structures are obliterated and their walls are thickened.
结论：患者供应皮质下结构的长穿动脉或分支动脉结构紊乱，血管壁增厚。
Conclusions FA values decrease in the different area of white matter in CADASIL. The white matter lesions in some regions result in cognitive impairment.
结论CADASIL患者不同脑白质区域的FA值显著下降，部分区域的白质损害与患者的认知功能减退有关。
As one of the causes of stroke, dementia and migraine, cerebral autosomal dominant with subcortical infarcts and leukoencephalopathy (CADASIL) has been paid increasing attention.
作为卒中、痴呆和偏头痛的病因，伴有皮质下梗死和白质脑病的常染色体显性遗传性脑动脉病(CADASIL)越来越受到人们的重视。
As one of the causes of stroke, dementia and migraine, cerebral autosomal dominant with subcortical infarcts and leukoencephalopathy (CADASIL) has been paid increasing attention.
作为卒中、痴呆和偏头痛的病因，伴有皮质下梗死和白质脑病的常染色体显性遗传性脑动脉病(CADASIL)越来越受到人们的重视。
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