Background-PRKAG2 mutations cause glycogen-storage cardiomyopathy, ventricular preexcitation, and conduction system degeneration.
背景-PRKAG2突变引起糖原贮积型心肌病，心室预激和传导系统退化。
Damage to the central nervous system (CNS) is always followed by an irreversible axon degeneration of injured neurons.
成年哺乳动物中枢神经系统(CNS)损伤，常导致神经元死亡及轴突不可逆的退化。
Objective To localize the functional involvement of the central nervous system (CNS) in patients with subacute combined degeneration (SCD).
目的探讨亚急性联合变性（SCD）患者中枢神经系统（CNS）功能受累的部位。
Conclusion IGF-1 system was involved in the pathogenic mechanism of MND, the lack of neurotrophic support might be one of causes in leading to degeneration of spinal motor neurons.
结论提示IGF - 1系统参与了MND的发病机理，神经营养支持的缺乏是导致脊髓运动神经元变性的原因之一。
The main pathological change was the mucoid degeneration of intestinal epithelial cells, and multi-system organs were involved.
主要的病理变化是肠道上皮的黏液变性，并累及多系统器官的一般变性；
It is finally concluded that the two external fields exert a high temperature and weak degeneration effect on the charged particle system.
研究结果表明，带电粒子系统在双外场约束下具有高温弱简并效应。
Basic composition and principle of the FBG strain sensing system were analyzed in detail. According to engineering experience we obtained that FBG sensor was most likely degeneration.
本文首先介绍了光纤光栅的传感原理，并详细分析了光纤光栅应变传感系统的基本组成以及工作原理，根据工程应用的经验得出了光纤光栅传感器最容易出现性能蜕化现象。
Basic composition and principle of the FBG strain sensing system were analyzed in detail. According to engineering experience we obtained that FBG sensor was most likely degeneration.
本文首先介绍了光纤光栅的传感原理，并详细分析了光纤光栅应变传感系统的基本组成以及工作原理，根据工程应用的经验得出了光纤光栅传感器最容易出现性能蜕化现象。
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