Excess fat makes your body resistant to insulin.
过多的脂肪使身体对胰岛素产生抵抗力。
Plus, the resistant starch in potatoes help your body burn fat, too.
另外，马铃薯中所含的淀粉也能帮助身体燃烧脂肪。
Resistant starch in a meal is associated with less fat storage after that meal.
在一餐的反抗的淀粉与那一一餐后的比较不胖储藏有关。
But it can get rid of fat resistant to traditional methods of losing weight.
但它确实能摆脱对传统的减肥方法有抵抗力的脂肪。
Methods the changes in insulin sensitivity and the effects of chloroquine were observed in insulin-resistant (IR) rats induced by high-fat feeding.
方法在高脂肪饮食喂养法复制的胰岛素抵抗(IR)大鼠模型中，观察大鼠的胰岛素敏感性的变化以及氯喹对其影响。
At the end of the experiment, the high-fat diet group was divided into dietary induced obesity (DIO) and dietary induced obesity resistant (DIO-R) subgroups according to the final body weight.
实验结束时，根据体重将高脂实验组大鼠分为饮食诱导肥胖(DIO)和饮食诱导肥胖抵抗(DIO R)大鼠，比较各组相关指标的差异。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To investigate the changes of glucose metabolism, plasma resistin and adiponectin in insulin-resistant (IR) rats induced by high-fat diet and lipid infusion.
目的探讨高脂喂养和脂质灌注诱导的胰岛素抵抗（IR）大鼠糖代谢、血浆抵抗素、脂联素水平和肌肉抵抗素蛋白表达的变化。
Dr Katarina Kos, lead author of the research, added: "Leptin is produced in fat cells to regulate appetite, but the body becomes resistant to the effects of appetite reduction in obese patients."
研究的主要作者科斯卡塔琳博士补充说：“瘦素是脂肪细胞产生用来调节食欲的，但肥胖患者的身体抵抗这种减少食欲的作用。”
Objective to observe the change of endothelium-dependent vasodilatation function and to explore its mechanism in insulin-resistant (IR) rats induced by high fat feed.
目的探讨高脂饲料诱导胰岛素抵抗(IR)动物模型的内皮功能变化及其机制。
Conclusions: To make the model of high-fat diet induced obesity-prone and obesity-resistant rats is according with the biochemistry characteristic of high-fat diet in public.
结论：高脂饮食诱导的肥胖易感和肥胖抗性大鼠模型制备成功，其生化特征符合人群高脂饮食致肥胖的特点。
Conclusions: To make the model of high-fat diet induced obesity-prone and obesity-resistant rats is according with the biochemistry characteristic of high-fat diet in public.
结论：高脂饮食诱导的肥胖易感和肥胖抗性大鼠模型制备成功，其生化特征符合人群高脂饮食致肥胖的特点。
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