Excess fat makes your body resistant to insulin.
过多的脂肪使身体对胰岛素产生抵抗力。
Resistant starch in a meal is associated with less fat storage after that meal.
在一餐的反抗的淀粉与那一一餐后的比较不胖储藏有关。
Methods the changes in insulin sensitivity and the effects of chloroquine were observed in insulin-resistant (IR) rats induced by high-fat feeding.
方法在高脂肪饮食喂养法复制的胰岛素抵抗(IR)大鼠模型中，观察大鼠的胰岛素敏感性的变化以及氯喹对其影响。
At the end of the experiment, the high-fat diet group was divided into dietary induced obesity (DIO) and dietary induced obesity resistant (DIO-R) subgroups according to the final body weight.
实验结束时，根据体重将高脂实验组大鼠分为饮食诱导肥胖(DIO)和饮食诱导肥胖抵抗(DIO R)大鼠，比较各组相关指标的差异。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To investigate the changes of glucose metabolism, plasma resistin and adiponectin in insulin-resistant (IR) rats induced by high-fat diet and lipid infusion.
目的探讨高脂喂养和脂质灌注诱导的胰岛素抵抗（IR）大鼠糖代谢、血浆抵抗素、脂联素水平和肌肉抵抗素蛋白表达的变化。
Dr Katarina Kos, lead author of the research, added: "Leptin is produced in fat cells to regulate appetite, but the body becomes resistant to the effects of appetite reduction in obese patients."
研究的主要作者科斯卡塔琳博士补充说：“瘦素是脂肪细胞产生用来调节食欲的，但肥胖患者的身体抵抗这种减少食欲的作用。”
Objective to observe the change of endothelium-dependent vasodilatation function and to explore its mechanism in insulin-resistant (IR) rats induced by high fat feed.
目的探讨高脂饲料诱导胰岛素抵抗(IR)动物模型的内皮功能变化及其机制。
Conclusions: To make the model of high-fat diet induced obesity-prone and obesity-resistant rats is according with the biochemistry characteristic of high-fat diet in public.
结论：高脂饮食诱导的肥胖易感和肥胖抗性大鼠模型制备成功，其生化特征符合人群高脂饮食致肥胖的特点。
Conclusions: To make the model of high-fat diet induced obesity-prone and obesity-resistant rats is according with the biochemistry characteristic of high-fat diet in public.
结论：高脂饮食诱导的肥胖易感和肥胖抗性大鼠模型制备成功，其生化特征符合人群高脂饮食致肥胖的特点。
应用推荐