The expression of PI3K protein was located in cytoplasm and (or) nucleus.
PI 3k蛋白表达主要定位于细胞质和(或)胞核。
So the study of PI3K-Akt maybe a new target in searching drug treatment of tumor.
因此，通过对PI3K通路的研究有望寻求肿瘤药物治疗的新靶点。
However, interfering with PI3K signaling might increase the risk of bacterial infections in humans.
但是干扰PI 3 K信号通路可能会增加患者细菌感染的机会。
Objective To investigate the roles of PI3K pathway in multicellular resistance of lung adenocarcinoma A549 cells.
目的探讨PI3K信号通路在肿瘤群集耐药中的作用。
Previous data showed that NOK could activate the phosphatidylinositol 3 -kinase (PI3K) pathway in stable BaF3 cells.
前期的研究表明，NOK在小鼠前B细胞(BaF3)中能够激活磷脂酰肌醇3-激酶(PI3K)信号通路。
They also contend in their patent application that suppressing PI3K will increase feeding in farmed animals and reduce feed waste.
他们在专利中还主张抑制PI3K还可以提高饲养动物的摄食量和减少食物浪费。
Conclusion the PI3K signal pathway may be involved in AHR and the airway remodeling through promoting airway smooth muscle cells proliferation in asthma.
结论PI 3 K信号通路可能通过促进气道平滑肌细胞增殖参与哮喘气道高反应性及气道重构过程。
The PI3K deltainhibitor idelalisib (Zydelig) and the BTK inhibitor ibrutinib (Imbruvica) have shown promise for patients with relapsed follicular lymphoma.
PI3K抑制剂idelalisib和BTK抑制剂ibrutinib对复发的滤泡性淋巴瘤显示出很好的潜力。
The PI3K deltainhibitor idelalisib (Zydelig) and the BTK inhibitor ibrutinib (Imbruvica) have shown promise for patients with relapsed follicular lymphoma.
PI3K抑制剂idelalisib和BTK抑制剂ibrutinib对复发的滤泡性淋巴瘤显示出很好的潜力。
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