About one in 500 Americans is genetically deficient in these structures, called LDL receptors, and, as a result, develops astronomically high levels of LDL cholesterol.
大约每五百个美国人中就有一个人的这个结构有遗传缺陷，这就是为什么他们的LDL胆固醇含量会达到这样一个天文数字。
In LDL binding studies, this luminal elastin layer limited LDL penetration, whereas its absence at the branches resulted in extensive LDL binding.
在低密度脂蛋白集合的研究中，官腔的弹力蛋白限制低密度脂蛋白的渗透性，在弹力蛋白缺失的分叉处导致大量低密度脂蛋白粘集。
The standard plasma lipid profile, the size of LDL particles and the plasma level of circulating ox-LDL (4E6 antibody) were determined.
并测定标准血浆脂蛋白，低密度脂蛋白颗粒大小以及血浆中循环氧化低密度脂蛋白（4E6抗体）水平。
Conclusion: HGF can up-regulate expression of LDL receptor in mesangial cells and increase mesangial cells uptake of LDL, so HGF may play a role in lipid-induced kidney injury.
结论：HGF通过促进系膜细胞表达LDL受体，增加系膜细胞对LDL的摄取，可能参与了脂质肾损伤过程。
So, we are really back to the original findings of the trial, namely that more LDL lowering produced a better outcome than less LDL lowering.
因此，我们回到这一实验的最初发现，就是LDL降低的越多，临床效果越好。
Atherosclerotic plaques of cholesterol mainly from LDL, LDL-C can be used as evaluation of the prognosis of patients with stable angina objective material indicators.
动脉粥样硬化斑块中的胆固醇主要来自LDL,LDL - C可以作为评价稳定型心绞痛患者预后的客观物质指标。
They also suggest that using LDL-P values to gauge the adequacy of LDL lowering may be a more effective approach to optimize management of diabetic and other high-risk patients.
他们还建议使用低密度脂蛋白粒子数的值来衡量低密度脂蛋白降低情况可能是优化糖尿病和其他高风险的患者管理一个更有效的方法。
CONCLUSION: OX-LDL enhances the expression and activity of MMP-2 in HUVEC, which may provide a explanation for the phenomenon that OX-LDL induces atherosclerotic lesions and plaque rapture.
结论：OX - LDL能促进内皮细胞表达MMP - 2，这提示血管细胞外基质的降解在氧化脂蛋白诱发的动脉粥样硬化斑块产生和破裂机制中起着一定作用。
RESULTS the electrophoretic mobility of serum LDL was slowed and the levels of TC and TG of LDL were decreased in the treatment groups.
结果服用消淤片后，兔血清ldl电泳迁移速率减慢，其所含的TC和TG含量减少。
The results showed that LDL-VLDL-GGT and the ratio of LDL-VLDL-GGT/ GGT were significantly higher in PHC than LC and CH(P<0.01).
结果表明，原发性肝癌血清LDL-VLDL-GGT以及LDL-VLDL-GGT占血清总GGT的百分比显著高于肝硬化、慢性肝炎及非肝脏疾病组（P＜0．01）。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
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