Low GABA levels are thought to be associated with depression and anxiety disorders.
低gaba水平被认为与抑郁和焦虑有关。
Experimentally blocking GABA to the posterior hypothalamus engenders the same response.
对后下丘脑阻断GABA的实验也出现了同样的反应。
GABA typically dampens neural activity, and many drugs for treating anxiety disorders target its receptors.
GABA通常会抑制神经活性，而且许多治疗焦虑症药物的目标就是(作用于)它的受体。
Once a stroke kills a swath of brain cells, a neurotransmitter known as GABA impairs the surviving, apparently healthy, brain tissue.
当中风杀死一个带状区域的脑细胞后，一种叫伽马氨基丁酸（GABA）的神经传递素将破坏残存的，看起来还健康的脑组织。
GABA containing callosal neurons were also found.
同时发现含gaba的胼胝体神经元。
The children of Ramah and Gaba, six hundred twenty and one.
拉玛人，迦巴人共六百二十一名。
One GABA-IR positive nerve fiber could form synapse with 6-7 OHCs.
一根GABA - IR阳性的神经纤维，在外毛细胞区可形成6 ~ 7个终末。
Glutamic acid decarboxylase (GAD) is a major GABA synthesizing enzyme.
谷氨酸脱羧酶(GAD)是合成GABA的主要限速酶。
Objective: to synthesize tiagabine hydrochloride, a GABA uptake inhibitor.
目的：合成GABA摄取抑制剂盐酸噻加宾。
GABA, he says, is like the marshal that prevents the cells from being too active.
他说GABA行使信号整合作用，避免细胞过度活化。
The protocerebral bridge and the postcerebral bridge show GABA-like immunoreactivity.
前脑桥和脑后桥都显示GABA阳性反应。
Gabapentin is a new anti-epileptic agent whose chemical structure resembles that of GABA.
加巴喷丁是一种新型抗癫痫药，其化学结构与GABA相似。
The tritocerebrum and suboesophageal ganglion show obvious GABA and 5-HT immunoreactivity.
后脑和咽下神经节髓质区均呈现明显的GABA和5-HT阳性反应。
The researchers then re-scanned each participant's brain, looking specifically at GABA levels.
随后研究人员重新扫描每位参与者大脑，而且特别关注GABA的水平。
The results demonstrated that GABA immunoreactive cells were mainly located in the pancreatic islets.
结果发现GABA免疫反应阳性细胞主要分布于胰腺内分泌部（胰岛）。
The results showed that GABA receptor agonists increased rat's pt, and GABA receptor antagonists decreased pt.
结果表明，GABA受体激动剂可提高大鼠痛阈，而拮抗剂降低大鼠痛阈。
But yohimbine could not antagonize the central hypotensive action of GABA, while picrotoxin could entirely block it.
但是育亨宾不能阻断侧脑室注射GABA的中枢降压作用，而苦味毒则能完全阻断其降压效应。
But yoga might be able to help, in part by stimulating the cranial nerve and an important neurotransmitter called GABA.
但瑜珈能够通过刺激脑神经和一种名叫GABA的神经递质来缓解病情。
The message that GABA transmits is an inhibitory one: it tells the neurons that it contacts to slow down or stop firing.
GABA传递的是抑制信息：它告诉脑神经细胞放缓或停止兴奋的信息。
The results suggest that possibly there is a mechanism of negative feedback autoregulation of GABA uptake system in mouse stomach.
本研究结果提示，胃中可能存在一种GABA摄取系统的负反馈式的自身调节机制。
The transplanted GABA receptors are similar in biochemico-pharmacological properties with native GABA receptors from chicken brain.
移植GABA受体与天然鸡脑GABA受体的生化药理学性质基本一致。
Objective: to study the mechanism of GABA receptor change in organophosphates acute poisoning and to offer a basis for its treatment.
目的：探索有机磷酸酯类化合物急性中毒的-氨基丁酸能系统机理，为有机磷酸酯类化合物急性中毒的治疗提供理论依据。
Under TEM, GABA-IR positive efferent endings, negative efferent endings and negative afferent endings could be seen on the OHC bases.
免疫电镜发现在外毛细胞底部有GABA - IR阳性的传出神经末梢及阴性的传入、传出神经末梢。
Levels of the chemical - called GABA - rise and fall dramatically in brains of people who are good at learning sequences of movements.
学习动作顺序时，大脑中一种称为GABA的化学物质含量起伏巨大。
Results: GABA-IR structure of both groups was observed in amacrine cells, inner plexiform layer, ganglion cells and nerve filament layer.
结果：老年猫、青年猫GABA免疫反应阳性结构均见于无长突细胞、内网状层、神经节细胞和神经纤维层。
Preparation and application of GABA are concerned. Preparation of GABA by Lactobacillus brevis was studied in detail in this dissertation.
本文系统地研究了利用短乳杆菌生物法制备GABA的过程及相关工艺。
We know its expression correlates with a subset of GABA neurons. We use that gene as a kind of entry point to express various kinds of markers.
我们知道该基因的表达与GABA神经元的一个子集有关，利用该基因作为一种切入点来表达不同种类的标记。
Conclusion NMDA receptor antagonists can inhibit the reduction of Dopamine, and GABA receptor antagonists can promote the reduction of Dopamine.
结论NMDA受体拮抗剂可抑制多巴胺神经递质的减少，GABA受体拮抗剂可促进多巴胺神经递质的减少。
Conclusion NMDA receptor antagonists can inhibit the reduction of Dopamine, and GABA receptor antagonists can promote the reduction of Dopamine.
结论NMDA受体拮抗剂可抑制多巴胺神经递质的减少，GABA受体拮抗剂可促进多巴胺神经递质的减少。
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