• One path he and his colleagues have followed is gamma-aminobutyric acid (GABA).

    他和他的同事们已经跟踪了氨基丁酸(GABA)这一种方法。

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  • GABA containing callosal neurons were also found.

    同时发现含gaba的胼胝体神经元。

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  • For the yoga study, researchers looked at the brain's GABA levels.

    在瑜珈研究中,研究者们研究他们大脑的GABA水平。

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  • Glutamic acid decarboxylase (GAD) is a major GABA synthesizing enzyme.

    谷氨酸脱羧酶(GAD)是合成GABA的主要限速酶。

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  • Objective: to synthesize tiagabine hydrochloride, a GABA uptake inhibitor.

    目的:合成GABA摄取抑制剂盐酸噻加宾。

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  • Low GABA levels are thought to be associated with depression and anxiety disorders.

    低gaba水平被认为与抑郁和焦虑有关。

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  • GABA, he says, is like the marshal that prevents the cells from being too active.

    他说GABA行使信号整合作用,避免细胞过度活化。

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  • Experimentally blocking GABA to the posterior hypothalamus engenders the same response.

    对后下丘脑阻断GABA的实验也出现了同样的反应。

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  • Gabapentin is a new anti-epileptic agent whose chemical structure resembles that of GABA.

    加巴喷丁是一种新型抗癫痫药,其化学结构与GABA相似。

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  • The molecule blocked a gene that codes for a receptor for GABA, a brain signaling molecule.

    该分子抑制了产生氨基丁酸受体这种大脑内信号分子的基因片段。

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  • Many times, the active ingredient of such preparations is GABA, or gamma amino butyric acid.

    多次的此类的准备工作的有效成分是GABA或伽玛氨基酸丁。

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  • The researchers then re-scanned each participant's brain, looking specifically at GABA levels.

    随后研究人员重新扫描每位参与者大脑,而且特别关注GABA的水平。

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  • The results demonstrated that GABA immunoreactive cells were mainly located in the pancreatic islets.

    结果发现GABA免疫反应阳性细胞主要分布于胰腺内分泌部(胰岛)。

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  • ResultsAfter seizures, the number of GABA receptors and the expression of GAD model group than group.

    结果致痫大鼠发作后,脑组织GABA受体数量较对照组数量增加,GAD表达增强,凋亡细胞出现。

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  • GABA typically dampens neural activity, and many drugs for treating anxiety disorders target its receptors.

    GABA通常会抑制神经活性,而且许多治疗焦虑症药物的目标就是(作用于)它的受体。

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  • The results showed that GABA receptor agonists increased rat's pt, and GABA receptor antagonists decreased pt.

    结果表明,GABA受体激动剂可提高大鼠痛阈,而拮抗剂降低大鼠痛阈。

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  • But yohimbine could not antagonize the central hypotensive action of GABA, while picrotoxin could entirely block it.

    但是育亨宾不能阻断侧脑室注射GABA的中枢降压作用,而苦味毒则能完全阻断其降压效应。

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  • But yoga might be able to help, in part by stimulating the cranial nerve and an important neurotransmitter called GABA.

    但瑜珈能够通过刺激脑神经和一种名叫GABA的神经递质来缓解病情。

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  • The message that GABA transmits is an inhibitory one: it tells the neurons that it contacts to slow down or stop firing.

    GABA传递的是抑制信息:它告诉脑神经细胞放缓或停止兴奋的信息。

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  • Drugs like propofol act on these GABA a receptors, mimicking and enhancing the effects of GABA, which inhibits cellular activity.

    异丙酚一类的药物能够作用于这类接收GABA的神经末梢,通过模拟和增强GABA引起的效果,起到抑制细胞活性的作用。

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  • Levels of the chemical - called GABA - rise and fall dramatically in brains of people who are good at learning sequences of movements.

    学习动作顺序时,大脑中一种称为GABA的化学物质含量起伏巨大。

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  • The results suggest that possibly there is a mechanism of negative feedback autoregulation of GABA uptake system in mouse stomach.

    本研究结果提示,胃中可能存在一种GABA摄取系统的负反馈式的自身调节机制。

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  • The transplanted GABA receptors are similar in biochemico-pharmacological properties with native GABA receptors from chicken brain.

    移植GABA受体与天然鸡脑GABA受体的生化药理学性质基本一致。

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  • Once a stroke kills a swath of brain cells, a neurotransmitter known as GABA impairs the surviving, apparently healthy, brain tissue.

    当中风杀死一个带状区域的脑细胞后,一种叫伽马氨基丁酸(GABA)的神经传递素将破坏残存的,看起来还健康的脑组织。

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  • Under TEM, GABA-IR positive efferent endings, negative efferent endings and negative afferent endings could be seen on the OHC bases.

    免疫电镜发现在外毛细胞底部有GABA - IR阳性的传出神经末梢及阴性的传入、传出神经末梢。

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  • Preparation and application of GABA are concerned. Preparation of GABA by Lactobacillus brevis was studied in detail in this dissertation.

    本文系统地研究了利用短乳杆菌生物法制备GABA的过程及相关工艺。

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  • We know its expression correlates with a subset of GABA neurons. We use that gene as a kind of entry point to express various kinds of markers.

    我们知道该基因的表达与GABA神经元的一个子集有关,利用该基因作为一种切入点来表达不同种类的标记。

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  • Results show that GABA-IR structure of both groups was observed in amacrine cells, inner plexiform layer, ganglion cells and nerve filament layer.

    结果显示老年猫、青年猫gaba免疫反应阳性结构均见于无长突细胞、内网状层、神经节细胞和神经纤维层。

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  • Results show that GABA-IR structure of both groups was observed in amacrine cells, inner plexiform layer, ganglion cells and nerve filament layer.

    结果显示老年猫、青年猫gaba免疫反应阳性结构均见于无长突细胞、内网状层、神经节细胞和神经纤维层。

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