小纤维蛋白血栓可在脑、心脏、肺、肾以及其它器官的小动脉中形成，引起组织缺血。
Small fibrin thrombi can form in small arteries of brain, heart, lungs, kidneys, and other organs to produce ischemic tissue damage.
目的探讨实验性肺血栓栓塞症(pte)后血栓形成及其意义。
Objective To study the significance of thrombosis after experimental pulmonary thromboembolism (PTE).
结论慢性呼吸衰竭患者存在高凝状态，并容易导致肺小动脉血栓形成。
Conclusions The patients with chronic respiration failure were in a state of high coagulation and contributed to thrombin small pulmonary artery.
所有动物都作尸检，检查人造瓣膜有无坏损，房、室面人造瓣膜上有无血栓形成，解剖心、肺、肝、脾、肾、大脑等脏器有无栓塞现象。
The heart, lungs and peripheral organs including the liver, spleen and kidney were excised and examined grossly to determine if thromboembolic phenomenon occurred.
前言：肺动脉高压的基本病理改变有三个方面：肺血管收缩、肺血管重构和肺小血管内微血栓形成。
The basic pathological alteration of pulmonary hypertension involves three dependent elements:pulmonary vasoconstriction, pulmonary vascular remodeling and pulmonary microvascular thrombogenesis.
前言：肺动脉高压的基本病理改变有三个方面：肺血管收缩、肺血管重构和肺小血管内微血栓形成。
The basic pathological alteration of pulmonary hypertension involves three dependent elements:pulmonary vasoconstriction, pulmonary vascular remodeling and pulmonary microvascular thrombogenesis.
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