小纤维蛋白血栓可在脑、心脏、肺、肾以及其它器官的小动脉中形成，引起组织缺血。
Small fibrin thrombi can form in small arteries of brain, heart, lungs, kidneys, and other organs to produce ischemic tissue damage.
目的探讨实验性肺血栓栓塞症(pte)后血栓形成及其意义。
Objective To study the significance of thrombosis after experimental pulmonary thromboembolism (PTE).
结论慢性呼吸衰竭患者存在高凝状态，并容易导致肺小动脉血栓形成。
Conclusions The patients with chronic respiration failure were in a state of high coagulation and contributed to thrombin small pulmonary artery.
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