方法：用脂多糖(LPS)诱发DHBV感染雏鸭急性肝坏死，建立实验模型。
Methods: An acute hepatic failure model was established in ducklings infected DHBV with lipopolysaccharides (LPS).
上述结果说明此模型与免疫异常有着密切关系，是研究肝坏死免疫发生机理的较理想模型。
These results strongly suggest that the abnormal immunity involved in the liver cell necrosis in the model.
另外，VPA对消化系统、肝脏及血液系统均有潜在的毒性作用，甚至出现致死性肝坏死。
In addition, VPA has potential toxicity for digestive system, blood system, and liver, even lethiferous putrescence.
更专业的名称是急性肝坏死综合症（ahpns），已对东南亚对虾生产产生了显着的影响。
EMS, more technically known as Acute Hepatopancreatic Necrosis Syndrome (AHPNS), has had a significant impact on shrimp production in Southeast Asia.
各种有害因素所致的肝损伤可表现为肝坏死、脂肪肝、胆汁郁积、肝纤维化、肝硬化及肝癌等。
The liver injury being caused by various harmful factors were manifested as hepatic necrosis, fatty liver, cholestasis, liver fibrosis, liver cirrhosis and liver cancer and so on.
目的探讨白细胞介素（IL） 18 抗体干预，了解IL 18 抗体对急性免疫性肝坏死动物模型的影响。
Objective To explore the effect of interleukin-18(IL-18) interference on the acute immunized hepatonecrosis animal model.
在上市后监督后已报告的严重肝脏不良反应事件包括肝坏死、黄疸、伴有或不伴有黄疸的暴发性肝炎、肝衰竭。
Postmarketing surveillance has reported cases of severe hepatic reactions, including liver necrosis, jaundice, fulminant hepatitis with and without jaundice, and liver failure.
本文应用实验性肝坏死大白鼠研究人胎肝细胞生长刺激物质（HSS）、抗肿瘤坏死因子（TNF）的作用及其机理。
The effects of HSS in experimental liver necrosis in rat and the mechanism of its anti-TNF effect in TNF-induced liver necrosis have been studied in this paper .
结果表明，实验性肝坏死时，血清中TNF活性较对照组明显升高，HGF及PGE_1能保护肝脏，并降低血清中TNF活性。
A significant elevation of TNF activity was observed in the experimental liver necrosis group but not in the control. HGF and PGE_1 protected the liver and decreased serum TNF activity in rats.
本实验的目的是证实醋酸可引起肝局部坏死，并且作用强于无水乙醇。
Objective: To confirm acetic acid injection can cause local necrosis of liver and its effect beyond absolute ethanol.
肝动脉血栓形成是最可怕的一种肝移植术后并发症，会导致肝小叶广泛坏死、肝梗死和胆管狭窄。
Hepatic artery thrombosis is one of the most dreaded complications of liver transplant and can cause massive hepatic necrosis, hepatic infarcts and biliary strictures.
上市后监测报告发现了重度肝脏不良反应病例，包括肝脏坏死、黄疸、暴发性肝炎伴或不伴黄疸、肝衰竭等。
Postmarketing surveillance reports include cases of severe hepatic reactions, including liver necrosis, jaundice, fulminant hepatitis with and without jaundice, and liver failure.
四项指标与肝组织炎症坏死及纤维化程度均呈显著相关（P<0.05）。
There were significant correlation(P<0.05) between the four indexes and the degree of inflammation, necrosis and fibrosis.
肝硬化是肝细胞坏死后，肝纤维化和肝细胞再生所致。
Ongoing liver damage with liver cell necrosis followed by fibrosis and hepatocyte regeneration results in cirrhosis.
结论：肝组织细胞坏死和凋亡在T1WI和T2WI上有明显的信号差异表现。
Conclusion: There were significant different signal changes on conventional T1WI and T2WI on the groups of necrosis and apoptosis.
目的：在自身免疫性肝炎和病毒性肝炎引起的肝损伤发病过程中，肝细胞的坏死和凋亡是共同特点。
Objective Massive necrosis and apoptosis of liver cells is the common feature of different kinds of liver diseases, such as viral hepatitis, alcoholic hepatitis, autoimmune hepatitis, etc.
结论：拉米夫定治疗慢性乙型肝炎，可以明显降低血清hbv - DNA及转氨酶水平，延缓肝组织纤维化的进展，减轻肝组织的炎症坏死。
Conclusion: Lamivudine treatment could decrease the HBV-DNA and transaminase level, and delay the progression of liver fibrosis and lighten inflammation and necrosis of liver.
肝损伤是各种肝病共同的病理基础，其病理特点为肝细胞变性、坏死，甚至凋亡。
Liver injury is pathological basis of various hepatic diseases. A characteristic feature of the pathology is hepatocyte degeneration, necrosis, even apoptosis.
治疗骨坏死应以补益肝肾、行气活血为治则，以补益药配伍活血药进行加减应用。
In treating osteonecrosis we would use medicine with invigorating liver and kidney and activating blood circulation and taking invigorating medicine as the compatible one.
结果患者出现门静脉血栓形成，肝栓塞坏死。
Results The patient was found to have destructive damage of portal vein.
消融结束后沿针道切开牛肝，观测组织凝固性坏死形态和范围。
Ox livers were cut off along the needle tract and the size and shape of coagulation necrosis zones were observed and measured.
增强后动脉期肿块实性部分轻度增强，囊变坏死区无强化。本组2例均未见肝内外胆管扩张。
Solid part are enhanced lightly during arterial phase, the area of cystoid variation and necrosis are no enhanced. 2 cases have non-dilated bile duct of liver inside and outside.
同时肝组织经he染色，光镜下评估脂肪变性、炎症和坏死程度。
Meanwhile, the fatty degeneration, the degrees of inflammation and necrosis of liver samples were evaluated under the light microscope after he staining.
微波作用后肝组织坏死与温度分布及声像图变化有良好相关性。
The necrosis of liver tissue after microwave irradiation showed a good correlation with temperature distribution and change in ultrasonogram.
结论经肝动脉区域性栓塞原发性肝癌的病灶坏死速度较快。
Conclusion Regional hepatic artery embolization in primary liver tumor can speed up the necrosis rate.
而肝泡状棘球蚴病的液化坏死区表现不一，部分病灶的液化坏死区呈明显低信号。
The appearance of liquefied necrotic component of hepatic alveolar echinococcosis was variable on ADC map, some showed low diffusivity(signal intensity).
加碘化油tae治疗肝癌组织坏死区碘元素含量最高，残留癌组织区其次，癌周肝组织最低。
The iodine TAE group had shown that there was the highest iodine content in the necrotic tissues and secondary in the residual tumor tissues and the lowest in the liver tissues around the tumor.
与对照组比较，实验组大鼠肝细胞和肝窦内皮细胞变性和坏死程度较轻。
Compared with the control group, the degenerative and necrotic degree of hepatocyte and sinusoidal endothelial cells was lighter in the experiment group.
与对照组比较，实验组大鼠肝细胞和肝窦内皮细胞变性和坏死程度较轻。
Compared with the control group, the degenerative and necrotic degree of hepatocyte and sinusoidal endothelial cells was lighter in the experiment group.
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