• 许多研究表明非酶糖化葡萄糖自身氧化伴随着游离产生一个糖化氧化过程

    Many studies show that nonenzymatic glycation and glucose autoxidation which are accompanied by generation of free radicals is a process of glycation and oxidation.

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  • 第一阶段酵母菌发酵:酵母菌发酵阶段大分子分解分子,把淀粉分解成氧化酒精糖化作用。

    First stage: yeast fermentation: yeast fermentation stage will decompose large molecules into small ones, decompose starch into carbon dioxide and alcohol, also known as glycation.

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  • 评估指标包括体重指数血压糖化血红蛋白a1c血脂氧化血栓炎症标志物

    Assessments included: BMI, blood pressure, A1C, plasma lipids, and markers of oxidation, thrombosis, and inflammation.

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  • 糖化促进氧化2型DM病人发生AS重要发病机制之一

    Oxidation of LDL promoted by glycosylation is one of important pathogenesis of AS formation in DM2 patients.

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  • 相同储藏环境下粳米糖化氧化,粳米的霉变老化籼米快。

    Under the same storage conditions, round-grained non-glutinous rice was easier for saccharification and oxidation and had rapider rot and aging than long-grained non-glutinous rice.

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  • 目的探讨脂糖舒对2型糖尿病血清蛋白非酶糖化氧化反应抑制作用

    Objective To explore the inhibiting effects of Zhitangshu (ZTS) on non-enzyme glycosylation of serum protein and on peroxide of lipid in serum of the rats with type-2-diabetes.

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  • 本发明涉及一种使用氧化还原反应分析样品糖化蛋白方法其中可以获得高度可靠测定值

    A method of assaying a glycated protein in a sample with the use of REDOX reaction, in which highly reliable measurement can be obtained.

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  • 发明提供利用氧化还原反应糖化蛋白质测定方法测定精度及测定灵敏度优良。

    The present invention provides a method of measuring a glycated protein in a sample using a redox reaction, by which the glycated protein can be measured accurately with high sensitivity.

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  • 结论脂糖2型糖尿病模型大鼠不仅明显降糖降脂、改善胰岛素抵抗胰岛素敏感性作用,而且对其血浆蛋白糖化和脂质过氧化亦有显著的抑制作用。

    Conclusion ZTS has remarkable effect of lowering the insulin resistance and inhibiting the non-enzyme glycosylation of serum protein and the lipid-peroxidation in type-2-diabetes rats.

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  • 结论脂糖2型糖尿病模型大鼠不仅明显降糖降脂、改善胰岛素抵抗胰岛素敏感性作用,而且对其血浆蛋白糖化和脂质过氧化亦有显著的抑制作用。

    Conclusion ZTS has remarkable effect of lowering the insulin resistance and inhibiting the non-enzyme glycosylation of serum protein and the lipid-peroxidation in type-2-diabetes rats.

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