不幸的是，那些感染了多药抗药性肺结核病的病人不得不服用效力较弱，但毒性更强，价格更高的药物。

Unfortunately, those infected with MDR have to be treated with less effective, more poisonous and more costly drugs.

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我们假设多药抗药性相关蛋白-1介导的白三烯C4内位-外位转运是致血管系统动脉粥样硬化的重要机制。

We hypothesize that inside-outside transport of leukotriene C4 (LTC4) via MRP1 is a substantial proatherogenic mechanism in the vasculature.

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结论—这些研究结果表明多药抗药性相关蛋白-1和白三烯C4都有致动脉粥样硬化作用，因此两者都有可能成为抗动脉粥样硬化治疗的靶向物质。

Conclusions-: These findings indicate that MRP1 and LTC4 exert proatherosclerotic effects and that both MRP1 and LTC4 are potentially promising targets for atheroprotective therapy.

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