不幸的是，那些感染了多药抗药性肺结核病的病人不得不服用效力较弱，但毒性更强，价格更高的药物。
Unfortunately, those infected with MDR have to be treated with less effective, more poisonous and more costly drugs.
我们假设多药抗药性相关蛋白-1介导的白三烯C4内位-外位转运是致血管系统动脉粥样硬化的重要机制。
We hypothesize that inside-outside transport of leukotriene C4 (LTC4) via MRP1 is a substantial proatherogenic mechanism in the vasculature.
结论—这些研究结果表明多药抗药性相关蛋白-1和白三烯C4都有致动脉粥样硬化作用，因此两者都有可能成为抗动脉粥样硬化治疗的靶向物质。
Conclusions-: These findings indicate that MRP1 and LTC4 exert proatherosclerotic effects and that both MRP1 and LTC4 are potentially promising targets for atheroprotective therapy.
在后来一次到南非的时候，我去了一家住有很多抗药性肺结核患者和耐多药结核病患者的医院，这是一种治愈率不到50%的顽疾。
On a later trip to South Africa, I paid a visit to a hospital for patients with MDR-TB, multi-drug resistant tuberculosis, a disease with a cure rate of under 50%.
膜转运蛋白高表达导致多药抗药是白念珠菌抗药性产生的最重要机制之一。
The overexpression of membrane proteins functioning as drug efflux pumps is one of the main mechanisms for multidrug resistance of Candida albicans.
膜转运蛋白高表达导致多药抗药是白念珠菌抗药性产生的最重要机制之一。
The overexpression of membrane proteins functioning as drug efflux pumps is one of the main mechanisms for multidrug resistance of Candida albicans.
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