铜属有色金属，呈棕红色，是人体必需的微量元素。铜导电性和导热性良好，有光泽和延展性，可溶于硝酸、浓硫酸和有机酸中，加热易氧化成氧化铜。急性中毒主要见于吸入氧化铜或碳酸铜细粉尘或烟雾而引发金属烟尘热，其他则为经消化道或皮肤的误吸或误用中毒。
050411摘要 关键词： 铜中毒；病理变化；血液指标；雏鸡 [gap=2031]Key words: copper toxicity；lesion；blood parameter；chicken
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...脉畸形；铜针；血清铜；肝脏；铜中毒 [gap=2311]Key words: venous malformations;copper needles;Cu2+;liver;copper toxicosis ...
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特发性铜中毒 idiopathic copper toxicosis
和特发性铜中毒 idiopathic copper toxicosis
一起绵羊铜中毒的诊治 China Animal Health
The content of store copper in liver and in blood of geese was tended to increase with the ingestion of feed in high concentration copper,It could be a specific index for the diagnosis of copper poisoning.
肝铜,血铜含量随着饲料铜含量增加而增加,可作为诊断铜中毒症的特异性指标。
参考来源 - 实验性鹅铜中毒机理研究The expression of Ctr-1 gene mRNA in geese liver of different groups were studied by the method of RT-PCR at the end of test,to study the pathogenesis of copper intoxication in geese.
在试验结束时,采用RT-PCR方法对不同组鹅肝组织Ctr-1基因mRNA表达量进行研究,以探讨鹅发生铜中毒的机制。
参考来源 - 实验性鹅铜中毒机理研究·2,447,543篇论文数据，部分数据来源于NoteExpress
但目前仍未阐明铜中毒时出现MB的机制。
The pathogenesis of MBs in copper toxicosis is still unresolved.
所以对那些本来已经铜中毒的人来说就会是一个问题。
This can be a problem for people who already have high copper (which is toxic).
急性铜中毒对金鱼肝、肾细胞结构的这种损伤在解除毒物12天后未见明显恢复。
The liver and renal ultrastructure damnifications of acute copper poisoning goldfish had not evidently comeback by 12 days within innocuity water.
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