因此，肾脏的组织型血管增渗酶也有可能被败血症引发的发炎反应刺激而释放出来，并产生作用。

Therefore, it is possible that renal tissue kallikrein-bradykinin system is also released and activated by the stimulation of inflammation in sepsis.

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结果：新生儿败血症按发病机制分为早发型（占29.5%）、晚发型（占63.4%）及院内获得型（占7.1%）。

Results:According to the pathogenesis of neonatal sepsis the patients were divided into early-onset(29.5%), late-onset(63.4) and hospital acquired type(7.1%).

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任何一种休克后都可发生MODS，但以感染型最常见，器官衰竭是败血症性休克的典型特征之一。

MODS can follow any type of shock but is most common when infection is involved; organ failure is one of the defining features of septic shock.

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