仅从运动性心肌肥大的生物学机制方面，就诱导其发生的刺激因素及其信号转导通路、基因表达等几个方面加以综述。
In this article we only reviewed the biological mechanism of exercise induced cardiac hypertrophy by its inductive stimulus, pass way for signal transduction and genetic expression.
目的：探讨一次性力竭运动后大鼠心房肌蛋白质组的表达特征，初步筛选对力竭运动应激有意义的心房肌蛋白质。
Objective to study the expressive feature of atrial muscle proteome after a single bout of exhaustive exercise and primarily screen out some meaningful responsive proteins to exercise stress.
一次性游泳运动能够提高大鼠下丘脑瘦素受体的表达，运动可能促进血瘦素向下丘脑转运。
Acute aerobic exercise can up-regulate the expression of leptin-receptor in hypothalamus of rats, and exercise may promote the transportation of leptin from blood to cerebrospinal fluid.
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