结果黄芩苷可以明显改善大鼠脑缺血再灌注所致的行为学障碍，降低梗死率，降低脑组织含水量，同时可以降低脑内NO、NOS和MDA的含量，增加SOD含量。
The effects of baicalin on ischemia-induced barriers to behavior, rate of infarction, water content of brain tissue, contents of NO, NOS, MDA and SOD in brain tissues were observed.
其临床表现为：运动迟缓、僵直、静止性震颤等，并常常伴有认知障碍、精神症状及其它行为学改变。
Its clinical manifestations include bradykinesia, rigidity and tremor, usually companied with cognitive impairment, mental disorder and other behavioral changes.
结果行为学检测结果表明，血管性痴呆大鼠有明显的学习、记忆障碍，且该组大鼠海马组织乙酰胆碱含量持续降低。
Results The created rat vascular dementia model showed significant alteration in leaning and memory function, and obvious decreased content of acetylcholine in hippocampal tissues.
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