脾虚组10只，与实验组同期造模给予胃肠生物电检测，但不用任何药物；
Spleen deficiency group: 10, making models and gastrointestinal bioelectric detection at the same period with experiment group, but without any drug.
结果：IL - 12及IL - 18引起明显脾虚证的表现，与利血平制作脾虚动物模型组相似。
Results: IL-12 and IL-18 induced prominent spleen deficiency syndrome similar with that induced by reserpine.
结论脾气虚证发生有免疫相关基因组学基础，脾虚时机体免疫功能紊乱。
ConclusionThe genesis of Pi-Qi deficiency syndrome has its immune associated genomic basis, and the immune functions are disordered in patients with that syndrome.
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