动物实验的成功促使研究小组进行试管研究,以确定二甲胺四环素疗法能否影响感染HIV的人类T细胞的潜伏病毒。
The success with the animal model prompted the team to study in test tubes whether minocycline treatment affected latency in human t cells infected with HIV.
克莱·门特斯表示,二甲胺四环素能够降低T细胞的激活和增殖能力,这是从HIV感染者变成艾滋病患者所要经历的两个重要步骤。
According to Clements, minocycline reduces the ability of t cells to activate and proliferate, both steps crucial to HIV production and progression toward full blown AIDS.
队伍使用分子标志物发现二甲胺四环素非常选择性的阻断T细胞激活的特定专门的信息传导途径。
The team used molecular markers to discover that minocycline very selectively interrupts certain specific signaling pathways critical for T cell activation.
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