脂肪性肝病是多种原因引起的病变主体在肝小叶、以肝细胞脂肪变性和脂肪蓄积过多为主的临床病理综合征。
Fatty liver disease (FLD) is a clinicopathological syndrome caused by a variety of reasons. The main lesions are in hepatic lobule with steatohepatitis and accumulation of triglycerides in the liver.
结论实验性大鼠脂肪肝肝细胞发生脂肪变性，肝脂、血脂含量同时升高，并有肝细胞损伤。
Conclusion in the experiments, the high lipid food rats displayed the liver cells fat denaturalization, the blood lipid and liver lipid increased with liver cells damage.
了解h CV核心蛋白在诱发肝脂肪变性中的分子学机制，对丙型肝炎相关性脂肪肝的病因学，预防和治疗等方面具有极大的促进作用。
Understanding the molecule mechanisms of HCV core protein induction hepatic steatosis may contribute to elucidate the etiology, prevention and treatment of HCV hepatic diseases.
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