结论：循环氧化低密度脂蛋白水平的升高与主动脉瓣膜狭窄中的瓣膜组织纤维钙化重构有密切联系。
Conclusion: Increased level of circulating ox-LDL is associated with worse fibrocalcific remodelling of valvular tissue in AS.
由于瓣叶周期性弯曲而造成的胶原纤维损伤会诱发瓣叶撕裂和钙化，直至瓣膜失功。
Collagen fiber damage caused by cyclic leaflet bending may ultimately lead to the observed leaflet tearing, calcification up to failure of BPVs.
瘤组织由梭形细胞和胶原纤维束交织而成，可发生玻璃样变和钙化。
Tumor tissue from the spindle cells and collagen fiber bundles cutting through, glass-like degeneration and calcification.
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