产生LTP的生物学基础之一是海马神经元突触上的N -甲基- D -天门冬氨酸(NMDA)受体。
And the N-methyl-D-aspartate (NMDA) receptor on the synaptosome in hippocampus is one of the biological bases of LTP.
目的:利用N -甲基- D -天门冬氨酸(NMDA)损伤小脑颗粒神经元的模型,探讨IL - 6对神经损伤的保护作用,为临床上应用细胞因子防治脑损伤提供新思路。
Objective: by using a cellular model to acute damage to cultured cerebellar granule neurons induced by N-methyl-D-aspartate (NMDA) to explore neuroprotective effect of IL-6.
目的观察外周n -甲基- D -天门冬氨酸(NMDA)受体对福尔·马林外周注射所致长时程痛敏的作用。
Objective to investigate the role of peripheral N-methyl-D-aspartate (NMDA) receptors in the long-term hyperalgesia induced by peripheral injection of formalin in rats.
应用推荐