高血压大鼠病程4个月时视网膜毛细血管周细胞水肿变性，肾小球毛细血管上皮细胞病变较轻。
The pericytes exhibited edema and deterioration in retinal vessels at 4 months, epithelial changes of glomerular capillary were relatively slight.
结果缺血性肠炎的病理改变以肠壁充血、水肿、出血及变性坏死为主，伴不同程度的炎症反应。
Results The main pathological features of IC are hyperemia, edema, hemorrhage, degeneration and necrosis of bowel wall with various degrees of inflammatory reaction.
HE染色脑缺血造模后，缺血灶出现部位恒定，缺血灶内大量神经元变性坏死，出现脑水肿表现。
After he stain the ischemia focus was constant with a lot of neuron degeneration and necrosis, and the manifestations of cerebral edema were observed too.
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