脑缺血能激活内源性血管生成因子，使缺血脑组织血管密度增高，新生毛细血管形成。
Cerebral ischemia activates endogenous angiogenic factors, and increase microvessel density and form new capillaries in ischemic brain tissues.
肿瘤细胞释放可溶性的促血管生成因子诱导新生血管生成。
Tumor cells release soluble angiogenic factors inducing neovascularization, a process referred to as the "angiogenic switch".
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