试验的结果是神经元和海马状突起茎细胞都在磁效应下增加了繁殖频率，从而证明了猜测的正确性。
The test results revealed that neurons and stem cells in the hippocampus both responded positively to the treatment by increasing their proliferation rates.
PPAR治疗的主要效应是增加大脑的体积，保存胰岛素和IGF - II受体突起神经元，保持学习和记忆。
The major effects of the PPAR treatments were to increase brain size, preserve insulin and IGF-II receptor bearing neurons, and preserve learning and memory.
目的探讨内毒素预处理对大鼠前脑缺血再灌注后海马神经元的保护效应及其可能机制。
Objective To investigate the protective effect of endotoxin pretreatment on hippocampal neurons in rat forebrain following ischemia reperfusion and its possible mechanism.
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