已知tlr 2活化会促进抗原呈现细胞表面共同刺激分子的表现，但其调控机制及讯息传递路径仍不是非常清楚。
TLR2 is reported to enhance the expression of costimulatory molecules on the antigen presenting cell, however the regulatory mechanisms are still unclear.
培养过程中，树突细胞会抓住这些抗原，将之切成碎片，然后呈现在细胞表面。
During this process, the dendritic cells pick up the antigens, chop them up and present them on their surfaces.
结果陈株、H8205、R 22 3株病毒抗原，可以直接激活补体C_3，电泳后呈现典型降解峰，而ALC96与正常细胞抗原则均为阴性。
The results showed that Chen, H8205, R22 strains antigens can directly activate C_3, typical degradative peak was found after electrophoresis. But ALC96 and normal antigen were negative.
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