心力衰竭(CHF) 发展过程中, 心脏发生几何形状和心肌结构的改变称为心脏重构
心脏重构(cardiac remodeling)是心脏损伤或在血液动力学地应激反应时，因为分子和基因表达地变化，招致心脏地大小、形状和功用发生变化。
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Objective To determine the effects and mechanisms ofN-acetylcysteine (NAC) on cardiac remodeling in hypertensive rat. Methods Hypertension model was established by coarctation ofabdominal aorta artery.
目的探讨N-乙酰半胱氨酸(NAC)对高血压大鼠心脏重构的影响及其作用机理。
参考来源 - NIt's well known that left ventricular remodeling is a very important mechanism of CHF.
目前已明确心脏重构(ventricular，remodeling)是导致CHF发生发展的基本机制，并已经成为动物实验和临床研究的热点之一。
参考来源 - 慢性心力衰竭患者血清MMP·2,447,543篇论文数据，部分数据来源于NoteExpress
因此间质改建是心脏重构的重要病理基础。
Therefore, matrix remodeling is pathologic basis of myocardial remodeling.
心脏重构是指发生于心肌损伤之后的分子和细胞事件所导致的心脏各腔室形态、内径和功能改变。
Cardiac remodeling is defined as an alteration in anatomy, inter diameter and function of cardiac Chambers caused by a series of molecular and cellular events after injury to the heart.
房间隔缺损封堵术后早期即出现心脏正向重构，且这些变化的程度和时间不受患者接受封堵时的年龄影响。
Positive cardiac remodeling occurs after percutaneous closure of large atrial septal defect, irrespective of the magnitude and length of volume overload.
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